Search results for "Hepatocyte Nuclear Factor 3-alpha"

showing 4 items of 4 documents

The human liver fatty acid binding protein (FABP1) gene is activated by FOXA1 and PPARα; and repressed by C/EBPα: Implications in FABP1 down-regulati…

2013

Liver fatty acid binding protein (FABP1) prevents lipotoxicity of free fatty acids and regulates fatty acid trafficking and partition. Our objective is to investigate the transcription factors controlling the human FABP1 gene and their regulation in nonalcoholic fatty liver disease (NAFLD). Adenovirus-mediated expression of multiple transcription factors in HepG2 cells and cultured human hepatocytes demonstrated that FOXA1 and PPARα are among the most effective activators of human FABP1, whereas C/EBPα is a major dominant repressor. Moreover, FOXA1 and PPARα induced re-distribution of FABP1 protein and increased cytoplasmic expression. Reporter assays demonstrated that the major basal activ…

Hepatocyte Nuclear Factor 3-alphaMaleRepressorBiologyFatty Acid-Binding ProteinsFatty acid-binding proteinMiceTransactivationNon-alcoholic Fatty Liver DiseaseNonalcoholic fatty liver diseaseCCAAT-Enhancer-Binding Protein-alphamedicineAnimalsHumansPPAR alphaadipocyte protein 2Molecular BiologyTranscription factorCells Culturedchemistry.chemical_classificationFatty acidHep G2 CellsCell Biologymedicine.diseaseMolecular biologyFatty LiverMice Inbred C57BLLipotoxicitychemistrybiology.proteinProtein BindingBiochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids
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CREBBP/EP300 acetyltransferase inhibition disrupts FOXA1-bound enhancers to inhibit the proliferation of ER+ breast cancer cells.

2021

ABSTRACTTherapeutic targeting of the estrogen receptor (ER) is a clinically validated approach for estrogen receptor positive breast cancer (ER+ BC), but sustained response is limited by acquired resistance. Targeting the transcriptional coactivators required for estrogen receptor activity represents an alternative approach that is not subject to the same limitations as targeting estrogen receptor itself. In this report we demonstrate that the acetyltransferase activity of coactivator paralogs CREBBP/EP300 represents a promising therapeutic target in ER+ BC. Using the potent and selective inhibitor CPI-1612, we show that CREBBP/EP300 acetyltransferase inhibition potently suppresses in vitro…

Hepatocyte Nuclear Factor 3-alphaMultidisciplinaryReceptors EstrogenAcetyltransferasesCell Line TumorMCF-7 CellsHumansBreast NeoplasmsFemaleCREB-Binding ProteinE1A-Associated p300 ProteinCell ProliferationPloS one
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Identification of ELF3 as an early transcriptional regulator of human urothelium

2014

AbstractDespite major advances in high-throughput and computational modelling techniques, understanding of the mechanisms regulating tissue specification and differentiation in higher eukaryotes, particularly man, remains limited. Microarray technology has been explored exhaustively in recent years and several standard approaches have been established to analyse the resultant datasets on a genome-wide scale. Gene expression time series offer a valuable opportunity to define temporal hierarchies and gain insight into the regulatory relationships of biological processes. However, unless datasets are exactly synchronous, time points cannot be compared directly.Here we present a data-driven ana…

Hepatocyte Nuclear Factor 3-alphaTime seriesTime FactorsPPARγMicroarrayNormal Human UrotheliumComputational biologyBiologyReal-Time Polymerase Chain ReactionBioinformaticsProto-Oncogene ProteinsGene expressionElectric ImpedanceTranscriptional regulationHumansRNA Small InterferingGeneTranscription factorMolecular BiologyDNA PrimersGene knockdownProto-Oncogene Proteins c-etsReverse Transcriptase Polymerase Chain ReactionMicroarray analysis techniquesGene Expression Regulation DevelopmentalCell DifferentiationCell BiologyMicroarray AnalysisImmunohistochemistryELF3DNA-Binding ProteinsDifferentiationGene Knockdown TechniquesGene chip analysisGene expressionUrotheliumTranscription FactorsDevelopmental BiologyDevelopmental Biology
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Foxa1 reduces lipid accumulation in human hepatocytes and is down-regulated in nonalcoholic fatty liver.

2012

Triglyceride accumulation in nonalcoholic fatty liver (NAFL) results from unbalanced lipid metabolism which, in the liver, is controlled by several transcription factors. The Foxa subfamily of winged helix/forkhead box (Fox) transcription factors comprises three members which play important roles in controlling both metabolism and homeostasis through the regulation of multiple target genes in the liver, pancreas and adipose tissue. In the mouse liver, Foxa2 is repressed by insulin and mediates fasting responses. Unlike Foxa2 however, the role of Foxa1 in the liver has not yet been investigated in detail. In this study, we evaluate the role of Foxa1 in two human liver cell models, primary cu…

MaleGene Expressionlcsh:MedicineBiochemistrychemistry.chemical_compoundNon-alcoholic Fatty Liver DiseaseMolecular Cell Biologylcsh:ScienceCells Culturedchemistry.chemical_classificationMultidisciplinaryLiver DiseasesFatty liverAnimal ModelsHep G2 CellsPeroxisomeMiddle AgedLipidsMedicineFemaleResearch ArticleAdultHepatocyte Nuclear Factor 3-alphamedicine.medical_specialtyPrimary Cell CultureDown-RegulationGastroenterology and HepatologyBiologyYoung AdultInsulin resistanceModel OrganismsInternal medicinemedicineAnimalsHumansBiologyAgedTriglyceridelcsh:RFatty acidProteinsLipid metabolismmedicine.diseaseLipid MetabolismRatsFatty LiverEndocrinologyMetabolismchemistryHepatocyteslcsh:QFOXA2SteatosisPLoS ONE
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